Lasting Night

Lasting night describes the extended parasympathetic state (vagotonia) that begins after conflictolysis, when the organism releases its fixation (in its psyche) on the conflict content and the special biological program enters the healing phase.

Just as lasting day characterizes the conflict-active phase, lasting night characterizes the healing phase.

In normotonia, vagotonia begins around biological night (4pm), when the organism naturally shifts toward rest, digestion, warmth, and repair. In the healing phase, this state is intensified. One’s metabolism behaves as though it is in an extended biological night cycle, prioritizing tissue restoration, fluid accumulation, warmth, appetite, and recovery.

The hallmarks of lasting night include difficulty falling asleep until after midnight, prolonged sleep, difficulty waking in the morning, increased appetite, reduced thirst, warm hands and feet, fever, inflammation, heaviness, fogginess, and a general lack of interest in the former conflict content. The organism may feel unusually tired or unmotivated, not because the conflict is still active, but because the individual is devoting energy to repair.

During lasting night, the tissue and functional changes from the conflict-active phase begin to reverse or restore:

• tumours break down (with microbial activity, if available),
• ulcerated or necrotic tissues rebuild (in new mesoderm tissues, usually to a larger size than before), and
• function is regained,

depending on the germ layer and SBS involved. The greater the conflict mass accumulated during the conflict-active phase, the more pronounced the lasting night experience will be.

The duration of lasting night depends on the particular special biological program more than on the conflict mass. New-brain tissues, in particular, are “on a schedule” and the duration of lasting night is relatively fixed unless there are complications such as kidney tubule syndrome or conflict relapses.

Lasting night is most strongly associated with PCL A, the edema phase. During PCL A, fluid gathers in the relevant brain relay and corresponding tissue as part of the repair process. This phase continues until the epi-crisis, which briefly brings a surge of sympathecotonia in order to squeeze edema from the brain relay and move the organism toward PCL B. After the epi-crisis, lasting night gradually diminishes as PCL B completes and the organism establishes a new normotonia.